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The program was reviewed by Dashun Oliva, last updated on January 15th, 2014Localized subendocardial myocardial perfusion reserve in hibernating versus non-hibernating myocardium.
In the setting of hibernating myocardium, myocardial ischemia has been shown to elicit hibernating myocardium, which can also lead to myocardial infarction. A subendocardial phenomenon has been described in the setting of coronary artery disease, where local reduction in blood flow alters myocardial metabolism and contractile function. We sought to compare subendocardial perfusion reserve between patients with hibernating myocardium and patients without hibernating myocardium. One hundred consecutive patients (age, 58±11 years; 57% men) who underwent first-pass and adenosine stress magnetic resonance imaging of the left ventricle with regadenoson were included. Regional myocardial blood flow was calculated in a short-axis segment-based fashion. Myocardial perfusion reserve was defined as the ratio between stress and rest myocardial blood flow. At rest, stress myocardial blood flow was significantly reduced in patients with hibernating myocardium compared to patients without hibernating myocardium (mean, 2.0±0.3 vs. 2.8±0.5 mL/g/min; P=0.002). In contrast, myocardial perfusion reserve was not significantly different between the two groups (1.7±0.3 vs. 2.2±0.4; P=0.07). Moreover, subendocardial myocardial perfusion reserve was significantly reduced in patients with hibernating myocardium compared to patients without hibernating myocardium (1.4±0.3 vs. 2.2±0.4; P// Copyright 2016 The etcd Authors
//
// Licensed under the Apache License, Version 2.0 (the “License

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